Therefore, it truly is conceivable which the JIP1/3 → ASK1 → MKK4 signaling module may serve as a conduit to activate JNK when endosomal trafficking is disrupted and glucose concentrations slide during the early levels of methuosis. Our obtaining that MKK4 was robustly activated because of the methuosis-inducing IPPs, MOMIPP https://mortonh296wbe9.mdkblog.com/profile